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Our findings demonstrate significant reductions in mortality, MAKE, and MALO across diverse patient populations, suggesting that this therapeutic approach could represent a paradigm shift in MASLD management

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Conclusions Subclinical studies and epidemiological observational studies indicated that exposure to dioxins as persistent endocrine-disrupting chemicals is a cause of metabolic dysfunction associated with steatotic liver disease, as evidenced by hepatic steatosis and the presence of obesity, type 2 diabetes, and abnormal lipid profiles, which is due to the activation of hepatic aryl hydrocarbon receptor

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